Covid Vaccines Damage All Hearts, Study Finds

new scientific study by Nakahara et al. tested COVID-vaccinated people to see if they have “silent” changes in heart muscle function that standard radiology tests could detect. The study shows very unsettling results.

Scientists measured myocardial 18Fluorine-fluorodeoxyglucose (18F-FDG) uptake. F-FDG has molecular similarity to glucose. However, 18F-FDG does not metabolize like glucose. Therefore, PET scans could detect it, and its presence shows the heart muscle’s abnormally high demand for glucose, indicative of abnormal cardiac function. More about it here.

Conclusions: Focal myocardial 18F-FDG uptake seen on oncologic PET/CT indicates a significantly increased risk for multiple myocardial abnormalities.

Indeed, this is what the Nakahara study finds:


The study included 303 nonvaccinated patients (mean age, 52.9 years; 157 females) and 700 vaccinated patients (mean age, 56.8 years; 344 females). Vaccinated patients had overall higher myocardial FDG uptake compared to nonvaccinated patients (median SUVmax, 4.8 vs median SUVmax, 3.3 ; P < .0001). Myocardial SUVmax was higher in vaccinated patients regardless of sex (median range, 4.7-4.9 ) or patient age (median range, 4.7-5.6) compared to corresponding nonvaccinated groups (sex median range, 3.2-3.9; age median range, 3.3-3.3; P range, <.001-.015). Furthermore, increased myocardial FDG uptake was observed in patients imaged 1-30, 31-60, 61-120, and 121-180 days after their second vaccination (median SUVmax range, 4.6-5.1) and increased ipsilateral axillary uptake was observed in patients imaged 1-30, 31-60, 61-120 days after their 2nd vaccination (median SUVmax range, 1.5-2.0) compared to the nonvaccinated patients (P range, <.001-<.001).

This was not supposed to happen! The COVID vaccine is not supposed to affect the heart in any way. It was promised to “stay in the arm.”

The explosive findings of the study are discussed in the editorial that the editor of the magazine, Dr. Bluemke, felt obliged to publish.

Dr. Bluemke’s editorial is somewhat apologetic, and he gives faint praise for COVID vaccines.

The development of messenger RNA (mRNA) COVID-19 vaccines is a remarkable biotech story. While traditional vaccines took 5-10 years to develop, the COVID-19 vaccines took less than a year. By comparison, the fastest conventional vaccine previously developed was the mumps vaccine, on a timescale of 4 years.

Dr. Bluemke also does not mince words. He explains that the findings are not due to chance:

The main results: asymptomatic patients vaccinated for COVID-19 before PET had about 40% greater radiotracer activity in the myocardium than unvaccinated individuals. The P value was low, less than .0001. This translates to only 1 time out of 10 000 that these results would occur by chance.

The editorial states that there is no rational way to ignore and explain away the negative findings of myocardial inflammation by Takahara et al.:

Vaccine manufacturers are aware of the adverse effects of mRNA vaccines. These adverse effects lead to vaccine hesitancy. The study results by Nakahara et al suggest that mild asymptomatic myocardial inflammation could be more common than we ever expected. This in turn would support a hypothesis of more severe systemic inflammation related to mRNA vaccination in some patients who present with symptoma-c myocarditis.

Dr. Bluemke calls for further research into this:

The investigators understood their first result was only the starting point. They next performed extensive “sensitivity analyses” – i.e., looking at the same data from multiple different directions. What if we account for age differences between groups, and the number of vaccinations? If mRNA vaccinations do cause asymptomatic myocardial inflammation, wouldn’t the effect be more likely shortly after vaccination, rather than 6 months later? Many of us who had COVID-19 vaccines had flu-like symptoms immediately after vaccination – perhaps those of us with common flu-like reactions would have more myocardial inflammation as well? Could trained readers see the differences visually? Or were the differences seen only after placing regions of interest on the heart that could be accidentally mispositioned? The list goes on. Great researchers are also skeptics – they need to prove the results to themselves.

Was the increase in myocardial inflammation due to a few unlucky patients driving high averages, with most people remaining unaffected?

Read the Whole Article


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